In the nervous system of adult mammals, the sectioning of a nerve is a dramatic event leading to an irreversible loss of irreversible function, because in adults, neurons have lost the regenerative properties they originally had in the embryo. The molecule developed by Neurophoenix has the remarkable property of preventing the destruction of the neuron and restoring the regeneration circuits. Following treatment with our molecule in animal models of optic neuropathy, the injured nerve not only survives but becomes able to stimulate the regrowth of the nerve fiber.
The mechanisms of action is particularly robust because of evolutionary selection of a key protein in a neurotropic virus. The propagation, multiplication and, ultimately, transmission of this virus which infects the nervous system of mammals depends on the virus’s ability to keep neurons alive following infection.